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For 10 years I have heard that X vitamin or Y remedy would lessen my extreme Asian glow.

Rebecca R, 29 - Orinda, CA
For 10 years I have heard that X vitamin or Y remedy would lessen my Asian glow...
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NoGlo® is backed by an unconditional 60-day money back guarantee. If you are not satisfied with NoGlo® for any reason, simply send the bottles back to us, for a full product cost refund.

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NoGlo® Ingredients


NoGlo® is composed of beneficial vitamins, anti-oxidants and amino acids. NoGlo’s unique formula has specific concentrations that are based upon years of peer-reviewed research and is fully based on nutrition, biochemistry and research. Here is what makes up NoGlo®‘s patented formulation::

Alpha Lipoic Acid: A powerful anti-oxidant, alpha lipoic acid also regenerates other anti-oxidants, like glutathione. Consuming alcohol lowers the amount of glutathione in your body because it reacts with the ethanol byproduct: acetaldehyde. The more glutathione available, the more protection you have. When drinking, the body is exposed to a lot of oxidative damage. Alpha lipoic acid will help protect you from this damage, and will maintain high levels of glutathione, which is a sulfur containing anti-oxidant that stops the oxidation that would otherwise be done by the toxic aldehyde. Supplementation with Alpha Lipoic Acid in healthy volunteers has demonstrated decreased oxidative stress on the body. This was shown without any side effects, even in very large dosages of the compound.

N-Acetyl Cysteine: This is a more active from of L-cysteine, an amino acid. N-Acetyl Cysteine will help the body generate glutathione, an important antioxidant that will help protect from damage, and help enzyme activity. N-Acetyl Cysteine will also help raise levels of other compounds required for positive health, like coenzyme A, that are important for alcohol metabolism.

Nicotinamide: Nicotinamide helps build up levels of a necessary compound needed for the alcohol metabolizing enzymes. A form of Vitamin B3 like Niacin, Nicotinamide is the least toxic. Niacin and nicotinamide are also needed for regular cellular metabolism and energy production. In order to have acetaldehyde dehydrogenase functioning at a high level, high concentrations of nicotinamide derivatives are necessary.

Pantothenic Acid: Pantothenic acid is also known as Vitamin B5. Coenzyme A is a compound required for the complete metabolism of alcohol and for the production of energy within the cell, so we can get value out of our food. Pantothenic acid is a necessary building block for Coenzyme A.

Vitamin C: Vitamin C is an important antioxidant and serves many biological functions within the body that are necessary for maintaining health. In addition, Vitamin C helps protect other anti-oxidants so that they can function optimally.

Vitamin A: A necessary vitamin, Vitamin A is also an anti-oxidant. It will also help to slow the accumulation of acetaldehyde in the blood by interacting with alcohol dehydrogenase, another ket alcohol enzyme. In addition, Vitamin A helps maintain general health and eyesight.

Thiamine: Also known as Vitamin B1, thiamine helps alpha lipoic acid function optimally. Thiamine is necessary for maintaining neurological function. It is also needed for proper catabolism of sugars and amino acids.

If you have any further scientific inquiries, please email us at support@GoNoGlo.com

Medical journal and publication sources:

1. Ames, Bruce, et al. High-dose vitamin therapy stimulates variant enzymes with decreased coenzyme binding affinity (increased Km): relevance to genetic disease and polymorphisms. AMERICAN JOURNAL OF CLINICAL NUTRITION. Volume 75, Issue 4.

2. Bedino, Sefano, et al. Initial Characterization of Aldehyde Dehydrogenase from Rat Testis Cytosol. Bilogical Chemistry. Volume 371. 1989.

3. Brenner, Charles. On the Non-Specific Degredation of Nicotinamide Riboside. Journal of Biological Chemistry. Volume 286, Issue 20. 2011.

4. Brooks, PJ. THE ROLE OF ACETALDEHYDE-DNA ADDUCTS IN ALCOHOL MEDIATED CARCINOGENESIS. Volume 34, Issue 8. 2010.

5. Brooks PJ, Enoch MA, Goldman D, Li TK, Yokoyama A. The alcohol flushing response: an unrecognized risk factor for esophageal cancer from alcohol consumption. PLoS Med 2009;6:e50.

6. Chao YC, Liou SR, Chung YY, Tang HS, Hsu CT, Li TK, et al. Polymorphism of alcohol and aldehyde dehydrogenase genes and alcoholic cirrhosis in Chinese patients. Hepatology 1994;19:360–6.

7. De Flora, Silvio, et al. In vivo effects of N-acetylcysteine on glutathione metabolism and on the biotransformation of carcinogenic and/or mutagenic compounds. Carcinogenesis. Volume 6, Issue 12. 1985.

8. Farres, Jaume, Wang, Xinping, et al. Effects of Changing Glutamate487 to Lysine in Rat and Human Liver Mitochondrial Aldehyde Dehydrogena. The Journal of Biological Chemistry. Volume 269, Issue 19. 1994.

9. Johnston, Carol, et al. Vitamin C Elevates Red Blood Cell Glutathione in Healthy Adults. AMERICAN JOURNAL OF CLINICAL NUTRITION. Volume 58, Issue 1. 1993.

10. Kamino K, Nagasaka K, Imagawa M, et al. Deficiency in mito-chondrial aldehyde dehydrogenase increases the risk for late-onset Alzheimer’s disease in the Japanese population. Biochemistry and Biophysics Research Community. Volume 273. 2000.

11. Langeland, Bjorn, et al. Metal binding properties of thiols; complexes with horse liver alcohol dehydrogenase. Comparative Biochemistry and Physiology. Volume 123. 1998.

12. Lee, Ki-Hwan, et al. Chaperonin GroESL mediates the protein folding of human liver mitochondrial aldehyde dehydrogenase in Escherichia coli. Biochemical and Biophysical Research Communications. Volume 298. 2002.

13. Lioret, C. Moyse, A. Acid metabolism: The citric acid cycle and other cycles. Comparative biochemistry A Comprehensive treatise. Volume V. 1963.

14. Majamaa, Kari, et al. Increase of Blood NAD+ and Attenuation of Lacticacidemia During Nicotinamide Treatment of a Patient with the MELAS Syndrome. Life Sciences. Volume 58, Issue 8. 1996.

15. Oze, Isao, et al. Comparison between self-reported facial flushing after alcohol consumption and ALDH2 Glu504Lys polymorphism for risk of upper aerodigestive tract cancer in a Japanese population. Volume 101, Issue 8. 2010.

16. Perez-Miller, S. Hina, Y. Ram, V. et al. Alda-1 is an agonist and chemical chaperone for the common human aldehyde dehydrogenase 2 variant. NATURE STRUCTURAL & MOLECULAR BIOLOGY. Volume 17, Issue 2. 2010

17. Suh, Jung, et al. (R)-a-Lipoic acid reverses the age-related loss in GSH redox status in post-mitotic tissues: evidence for increased cysteine requirement for zGSH synthesis. Biochemistry and Biophysics. Volume 423. 2003.

18. Wang, Xinping. Heterotetramers of Human Liver Mitochondrial (Class 2) Aldehyde Dehydrogenase Expressed in Escherichia coli. Journal of Biological Chemistry. Volume 271, Issue 49. 1996.

19. Weatherman, R. Crabb, D. Alcohol and Medication Interactions. Alcohol and Research Health. Volume 23, Issue 1. 1999.

20. Xiao, Qing. The Mutation in the Mitochondrial Aldehyde Dehydrogenase (ALDH2) Gene Responsible for Alcohol-induced Flushing Increases Turnover of the Enzyme Tetramers in a Dominant Fashion. Journal of Clinical Investigation. Volume 98, Issue 9. 1996.

21. Yokoyama A, et al. Alcohol and Aldehyde Dehydrogenase Polymorphisms and a New Strategy for Prevention and Screening for Cancer in the Upper Aerodigestive Tract in East Asians. Keio Journal of Medicine. Volume 59, Issue 4. 2010.

22. Yu, Peter et al. Cutaneous Vasomotor Sensitivity to Ethanol and Acetaldehyde: Subtypes of Alcohol-Flushing Response Among Chinese. Alcoholism: Clinical and Experimental Research. Volume 14, Issue 6. 2006.

23. Chou, CF et al. Kinetic Mechanism of Human Class IV Alcohol Dehydrogenase Functioning as Retinol Dehydrogenase. Biological Chemistry. Volume 28, Issue 227. 2002.

24. Borcea V, Nourooz-Zadeh J, Wol SP, et al. alpha-Lipoic acid decreases oxidative stress even in diabetic patients with poor glycemic control and albuminuria. Free Radic Biol Med. 1999;26:1495–1500.

25. Jialal, I, Singh, U. Alpha-lipoic acid supplementation and diabetes. NUTRITION REVIEWS. Volume: 66, Issue: 11. 2008.